Predictors of Cognitive Dysfunction After Subarachnoid Hemorrhage
نویسندگان
چکیده
منابع مشابه
Predictors of cognitive dysfunction after subarachnoid hemorrhage.
BACKGROUND Cognitive dysfunction is a common and disabling sequela of subarachnoid hemorrhage (SAH). Although several clinical and radiographic findings have been implicated in the pathogenesis of cognitive dysfunction after SAH, few prospective studies have comprehensively and simultaneously evaluated these risk factors. METHODS Between July 1996 and March 2000, we prospectively evaluated 11...
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BACKGROUND AND PURPOSE Subarachnoid hemorrhage (SAH) frequently results in myocardial necrosis with release of cardiac enzymes. Historically, this necrosis has been attributed to coronary artery disease, coronary vasospasm, or oxygen supply-demand mismatch. Experimental evidence, however, indicates that excessive release of norepinephrine from the myocardial sympathetic nerves is the most likel...
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The main consequence of subarachnoid hemorrhage, for those who survive bleeding, is delayed, persistent vasospasm of intracranial conduit arteries which occurs between the third and seventh day after the insult and results in symptomatic brain ischemia in about 40% of cases. This vasospasm is considered to be a major cause of disability of post-SAH patients. Despite extensive experimental and c...
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BACKGROUND Aneurysm rebleeding has historically been an important cause of mortality after subarachnoid hemorrhage (SAH). OBJECTIVE To describe the frequency and impact of rebleeding in the modern era of aneurysm care, which emphasizes early surgical or endovascular treatment. DESIGN Inception cohort. SETTING Tertiary care medical center. PATIENTS A total of 574 patients enrolled in the...
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A cross section through a mouse quadriceps nerve showing discontinuous Drp2 staining (green) in the plasma membrane of Schwann cells, with axons stained for neurofilament (blue) and myelin stained for P0 (red). For more information, see the article by Sherman et al. (pages 9419 –9428).
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ژورنال
عنوان ژورنال: Stroke
سال: 2002
ISSN: 0039-2499,1524-4628
DOI: 10.1161/hs0102.101080